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Anonymous File :-(, x)
>>39370
Well it turns out that nobody in this thread is fully right or fully wrong:
From wiki:
"During power-intensive exercises such as sprinting, when the rate of demand for energy is high, lactate is produced faster than the ability of the tissues to remove it and lactate concentration begins to rise. This is a beneficial process since the regeneration of NAD+ ensures that energy production is maintained and exercise can continue. The increased lactate produced can be removed in a number of ways including
* oxidation to pyruvate by well-oxygenated muscle cells which is then directly used to fuel the citric acid cycle and * conversion to glucose via the Cori cycle in the liver through the process of gluconeogenesis.
Contrary to popular belief, this increased concentration of lactate does not directly cause acidosis, nor is it responsible for delayed onset muscle soreness.[1] This is because lactate itself is not capable of releasing a proton, and secondly, the acidic form of lactate, lactic acid, cannot be formed under normal circumstances in human tissues. Analysis of the glycolytic pathway in humans indicates that there are not enough hydrogen ions present in the glycolytic intermediates to produce lactic or any other acid."
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